The term “skin cancer” has long been used to describe the painful and painful effects of aging and the harmful effects of pollution.
For many people, though, the term is more appropriate.
A new study published in the journal Cell Research suggests that cells that have been exposed to sunlight are more sensitive to UVB, the sun’s harmful rays.
“We were really interested in whether or not the sun protects us against UVB,” said study co-author Dr. Yael Gefen, a dermatologist at Tel Aviv University.
“This is a critical question in terms of how we can protect ourselves from sunburn.”
Gefen and her colleagues discovered that in the presence of sunlight, cells that had been exposed for a long time to UV rays are able to repair themselves, which is important for the immune system to function properly.
“There are several ways that sunlight protects the skin,” Gefens said.
“We can protect our skin from the sun, we can help our skin regenerate itself, we may be able to fight infections and skin cancer.”
The researchers found that cells with the same type of UV receptor that were exposed to UV light were more sensitive and more sensitive than cells that were not.
In addition, when exposed to the sun in combination with the chemical retinol, which helps prevent wrinkles, cells were more able to respond to UV radiation.
“This is an example of a new phenomenon, which we call the sunscreen effect, where the skin cells have the ability to repair itself,” Gegen said.
“That means that the cells have a higher capability to withstand UV exposure, which means they are more tolerant to UV.”
The study was published online on June 25 in the prestigious journal Cell.
The research was funded by the American Cancer Society, and the researchers are part of a broader study on the mechanisms by which UV radiation affects cells in the body.
According to Gefes research team, UV rays penetrate the skin in the form of ultraviolet radiation (UV), which is absorbed by the cells and triggers the production of enzymes that help the cells fight off UV radiation and protect themselves from UV radiation, a process known as retinoblastoma.
“When we look at retinogenesis, it’s the process by which retinoids help the skin to maintain normal cells,” Geren said.
Retinoblasts are specialized cells that help maintain the skin’s color and appearance.
“Our data suggest that UVB radiation, in addition to increasing the levels of retinoid production, is also responsible for the enhancement of these cells’ ability to resist UV,” Geren said.
The researchers are now working to understand how retinogen production can be triggered by UV exposure.
“If you want to get a good UV protection, you need to get UVB exposure,” Gega said.
However, they do not have any concrete evidence that this is the case yet.
“What we know for sure is that the higher the UV intensity, the higher is the resistance of retinal cells to retinogenic UVB-induced degradation of their proteins,” Gefer said.
For more on sun exposure, read this post: The Sun: The Real Truth About Exposure to the Sun, From Your Sunscreen to Your Skin Cancer.